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BOOK
Title Protooncogenes and growth factors in steroid hormone induced growth and differentiation / edited by Sohaib A. Khan and George M. Stancel.
Imprint Boca Raton : CRC Press, 1994.

Copies/Volumes

LOCATION CALL # STATUS
 Main Stacks  QP552.G76 P77 1994    AVAILABLE
Description 277 p. : ill. ; 25 cm.
Bibliog. Includes bibliographical references and index.
Subject Growth factors -- Physiological effect.
Proto-oncogenes.
Steroid hormones -- Physiological effect.
Steroid hormones -- Receptors.
Cellular signal transduction.
Cell differentiation.
Gene Expression Regulation.
Growth Substances -- physiology.
Proto-Oncogene Proteins -- genetics.
Proto-Oncogene Proteins -- physiology.
Receptors, Steroid -- physiology.
Contents Steroid receptors and their regulation by phosphorylation -- Growth factors as mediators of sex steroid hormone action in the uterus during its preparation for implantation -- Steroid hormone induction of growth factors and oncogene expression in the uterus -- Glucocorticoid inhibited production of paracrine and autocrine acting growth factors in growth suppressible epithelial tumor cells -- Nuclear protooncogenes as regulatory genes and as marker genes for steroid action in all target tissues -- Uterine c-fos: a paradigm for control of gene expression during estrogen stimulated target tissue growth -- The jun protooncogene family in uterine growth and differentiation -- Growth factor regulation of c-fos protooncogene transcription -- Steroid regulation of growth factors and protooncogenes in the normal and malignant mammary gland -- Estrogen, c-myc, and breast cancer -- Molecular genetics of human endometrial carcinoma.
The c-erbB-2 oncogene in human prostrate cancer growth and progression -- The role of glucocorticoids in cell growth, differentiation, and programmed cell death -- Contributions of cell structure to oncogene and steroid hormone-mediated transcriptional control of gene expression supporting osteoblast proliferation and differentiation.
Other Author Khan, Sohaib A.
Stancel, George M.